# Compressive Vasomotion

> Compressive Vasomotion Hypothesis (CVH) — vasomotion as a fast compression sweep that collapses ambivalent neural resonances (the Bayesian-blur problem) into a definite state. Use when modeling the ~100ms taṇhā 'grab', precision-weighting as compression forcefulness, or the generative collapse step of vasocomputational active inference.

- Skill: `plurigrid/compressive-vasomotion` (Agent Skill, multi-file: 2 files)
- Install (CLI): `npx skillmds@latest add plurigrid/compressive-vasomotion`
- Raw SKILL.md: https://api.skillmd.com/api/skills/plurigrid/compressive-vasomotion/raw
- Safety review: pending
- Works with: Claude Code, Claude.ai, OpenAI Codex
- Category: AI & ML
- License: MIT
- Author: plurigrid (https://skillmd.com/u/plurigrid)
- Updated: 2026-09-17
- Page: https://skillmd.com/skills/plurigrid/compressive-vasomotion

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# compressive-vasomotion

The **Compressive Vasomotion Hypothesis (CVH)** (Johnson 2023): the vasomotion reflex — the undulation of VSMCs wrapping every blood vessel — functions as a **compression sweep on nearby neural resonances**, collapsing and merging fragile, ambivalent patterns (the "Bayesian blur") into a more durable, definite state. This is the **+1 / generate** leg of the vasocomputation timescale triad: the fast initial "grab" that *creates* specificity.

## Use When

- Modeling the ~25–100 ms taṇhā "fast grabby thing" as a physical compression event
- Reframing precision-weighting as two terms: sensory clarity available vs. compression *forcefulness* applied (the KL divergence between them ≈ a number for taṇhā)
- Representing the generative collapse step where a superposition of interpretations is jostled into one
- Coupling local electromagnetic-field dissonance → vasomotion trigger → resonance collapse

## Core Concepts

- **Compression sweep**: motifs of vasomotion, reflexive reactions to uncertainty, and patterns of taṇhā are *equivalent*. The brain pushes "what is" toward stable, satisfactory, controllable — the three marks inverted.
- **Bayesian blur**: an ambivalent SOHM superposition that has not yet committed; CVH collapses it (cf. collapsing a probability distribution / pinching a critical network into a definite circuit).
- **Trigger**: VSMC contractions are expected to be triggered by *local dissonance in the electromagnetic field* and to act back on neurons via ephaptic coupling, reduced blood flow, and altered local resonance.
- **Right amount ≠ zero**: a finite brain *must* compress away patterns or drown in sensory chaos; the cost is metabolic + epistemic. CVH is unskillful only when over-applied.

## GF(3) Balanced Triad

```
compressive-vasomotion (+1) ⊗ vascular-clamp (0) ⊗ latched-hyperprior (−1) = 0 (mod 3)
```

**Skill Trit**: +1 (Play / generate — the sweep *produces* a candidate collapse; cf. match-fires in the PAM RETE).

## Concomitant Skills

| Skill | Trit | Interface |
|-------|------|-----------|
| `vascular-clamp` | 0 | downstream: freezes what the sweep collapsed |
| `latched-hyperprior` | −1 | downstream: cements a sustained collapse |
| `vasocomputation` | +1 | umbrella / vascular substrate |
| `kolmogorov-compression` | +1 | compression progress as a drive (Schmidhuber) |
| `fokker-planck-analyzer` | +1 | stationary collapse of a resonance landscape |
| `information-geometry` | 0 | precision-weighting / KL of clarity vs. grab |

## Current literature (2024–2026)

- **van Veluw et al. (2020), Neuron** — spontaneous ~0.1 Hz vasomotion drives paravascular clearance; amplitude is tunable.
- **Hauglund / Nedergaard et al. (2025), Cell** — locus-coeruleus NE infraslow oscillations drive slow vasomotion → glymphatic clearance in NREM; zolpidem suppresses it.
- **Kedarasetti & Drew (2024), Neuron** — vasomotion travels as **long-wavelength waves**; resting modulation exceeds evoked. Recasts the "compression sweep" as a literal traveling wave, not a global clock.
- **Atasoy et al. (2016), Nat Commun** — connectome harmonics; with Safron's SOHMs gives the "out-of-tune harmonic → contraction" claim a formal substrate.
- **Pinotsis & Miller (2023), Prog Neurobiol** — cytoelectric coupling: endogenous fields sculpt activity (grounds the ephaptic loop; VSMC→field causation still unestablished).
- **Sharpening**: specify *which* ~0.1 Hz band (myogenic vs LC/NE vs Mayer) drives compression — the 2025 literature warns these are conflated.
- **Hook**: vasomotion phase should predict bistable-percept switching; optogenetic VSMC constriction should narrow local neural dynamic range / lower LFP entropy.
- **Grounded**: vasomotion exists + glymphatic role + ephaptic fields. **Speculative**: vasomotion as a Bayesian-collapse mechanism.

## References

- Johnson, M.E. (2023). *Principles of Vasocomputation, Part I*. opentheory.net (§V, CVH).
- Schmidhuber, J. (2008). *Driven by Compression Progress*. arXiv.
- Safron, A. (2020). *IWMT*. Frontiers in AI 3. (SOHMs as autoencoders/symmetry detectors.)
- Carhart-Harris & Friston (2019). *REBUS and the Anarchic Brain*. Pharmacol. Rev. 71(3).